Publications by Bernt Christian Hellerud
29 publications found
Original articles
Neisseria meningitidis accumulate in large organs during meningococcal sepsis
Front Cell Infect Microbiol, 13, 1298360
DOI 10.3389/fcimb.2023.1298360, PubMed 38089821
Transcriptomic changes in the large organs in lethal meningococcal shock are reflected in a porcine shock model
Front Cell Infect Microbiol, 12, 908204
DOI 10.3389/fcimb.2022.908204, PubMed 36034711
Choice of immunoassay to evaluate porcine cytokine levels
Vet Immunol Immunopathol, 230, 110129
DOI 10.1016/j.vetimm.2020.110129, PubMed 33059181
Extensive Changes in Transcriptomic "Fingerprints" and Immunological Cells in the Large Organs of Patients Dying of Acute Septic Shock and Multiple Organ Failure Caused by Neisseria meningitidis
Front Cell Infect Microbiol, 10, 42
DOI 10.3389/fcimb.2020.00042, PubMed 32154187
Traceability and distribution of Neisseria meningitidis DNA in archived post mortem tissue samples from patients with systemic meningococcal disease
BMC Clin Pathol, 17, 10
DOI 10.1186/s12907-017-0049-9, PubMed 28824331
Combined inhibition of C5 and CD14 efficiently attenuated the inflammatory response in a porcine model of meningococcal sepsis
J Intensive Care, 5, 21
DOI 10.1186/s40560-017-0217-0, PubMed 28261486
Properdin binding to complement activating surfaces depends on initial C3b deposition
Proc Natl Acad Sci U S A, 114 (4), E534-E539
DOI 10.1073/pnas.1612385114, PubMed 28069958
Massive Organ Inflammation in Experimental and in Clinical Meningococcal Septic Shock
Shock, 44 (5), 458-69
DOI 10.1097/SHK.0000000000000441, PubMed 26473439
A complement C5 gene mutation, c.754G>A:p.A252T, is common in the Western Cape, South Africa and found to be homozygous in seven percent of Black African meningococcal disease cases
Mol Immunol, 64 (1), 170-6
DOI 10.1016/j.molimm.2014.11.010, PubMed 25534848
Systemic CD14 inhibition attenuates organ inflammation in porcine Escherichia coli sepsis
Infect Immun, 81 (9), 3173-81
DOI 10.1128/IAI.00390-13, PubMed 23774598
Combined inhibition of complement C5 and CD14 markedly attenuates inflammation, thrombogenicity, and hemodynamic changes in porcine sepsis
J Immunol, 191 (2), 819-27
DOI 10.4049/jimmunol.1201909, PubMed 23761634
Activation of coagulation and platelets by candidate membranes of implantable devices in a whole blood model without soluble anticoagulant
J Biomed Mater Res A, 101 (2), 575-81
DOI 10.1002/jbm.a.34348, PubMed 22949225
Polyvalent immunoglobulin significantly attenuated the formation of IL-1β in Escherichia coli-induced sepsis in pigs
Immunobiology, 218 (5), 683-9
DOI 10.1016/j.imbio.2012.08.268, PubMed 22947599
Inflammatory response induced by candidate biomaterials of an implantable microfabricated sensor
J Biomed Mater Res A, 100 (5), 1142-50
DOI 10.1002/jbm.a.34054, PubMed 22337626
The role of bradykinin and the effect of the bradykinin receptor antagonist icatibant in porcine sepsis
Shock, 36 (5), 517-23
DOI 10.1097/SHK.0b013e3182336a34, PubMed 21921836
Activation of polymorphonuclear leukocytes by candidate biomaterials for an implantable glucose sensor
J Diabetes Sci Technol, 5 (6), 1490-8
DOI 10.1177/193229681100500625, PubMed 22226271
Neisseria meningitidis and Escherichia coli are protected from leukocyte phagocytosis by binding to erythrocyte complement receptor 1 in human blood
Mol Immunol, 48 (15-16), 2159-69
DOI 10.1016/j.molimm.2011.07.011, PubMed 21839519
Complement activation by candidate biomaterials of an implantable microfabricated medical device
J Biomed Mater Res B Appl Biomater, 98 (2), 323-9
DOI 10.1002/jbm.b.31855, PubMed 21648060
Dissecting the effects of lipopolysaccharides from nonlipopolysaccharide molecules in experimental porcine meningococcal sepsis
Crit Care Med, 38 (6), 1467-74
DOI 10.1097/CCM.0b013e3181de8c94, PubMed 20400898
Critical roles of complement and antibodies in host defense mechanisms against Neisseria meningitidis as revealed by human complement genetic deficiencies
Infect Immun, 78 (2), 802-9
DOI 10.1128/IAI.01044-09, PubMed 19933829
CD14 inhibition efficiently attenuates early inflammatory and hemostatic responses in Escherichia coli sepsis in pigs
FASEB J, 24 (3), 712-22
DOI 10.1096/fj.09-140798, PubMed 19841036
A new dynamic porcine model of meningococcal shock
Shock, 32 (3), 302-9
DOI 10.1097/SHK.0b013e31819c37be, PubMed 19174740
Human genetic deficiencies reveal the roles of complement in the inflammatory network: lessons from nature
Proc Natl Acad Sci U S A, 106 (37), 15861-6
DOI 10.1073/pnas.0903613106, PubMed 19717455
Zip lock poly bags in drug cases-A valuable source for obtaining identifiable DNA results?
Forensic Sci. Int. Genet. Suppl. Ser., 1 (1), 433-434
DOI 10.1016/j.fsigss.2007.10.013
Stages of meningococcal sepsis simulated in vitro, with emphasis on complement and Toll-like receptor activation
Infect Immun, 76 (9), 4183-9
DOI 10.1128/IAI.00195-08, PubMed 18591229
New biomarkers in an acute model of live Escherichia coli-induced sepsis in pigs
Scand J Immunol, 68 (1), 75-84
DOI 10.1111/j.1365-3083.2008.02122.x, PubMed 18466195
[A three-year-old boy with hypoglycaemia]
Tidsskr Nor Laegeforen, 125 (15), 2031-2
PubMed 16114124
Skin conductance and behaviour during sensory stimulation of preterm and term infants
Early Hum Dev, 70 (1-2), 35-46
DOI 10.1016/s0378-3782(02)00070-1, PubMed 12441203
Theses
Meningococcal sepsis: beneficial and harmful inflammatory mechanisms
In Series of dissertations submitted to the Faculty of Medicine, University of Oslo, Unipub, Oslo, no. 1142, 1 b. (flere pag.)
BIBSYS 112822711, ISBN 978-82-8264-141-8